RT @agingdoc1: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induce…
Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication https://t.co/P0m4fwI0v3 @Oncotarget
New article: Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication https://t.co/VsOzhowbwn #AML #leusm #hem
#PaperoftheDay: "Pim kinase inhibitor co-treatment decreases alternative non-homologous end-joining DNA repair and genomic instability induced by topoisomerase 2 inhibitors in cells with FLT3 internal tandem duplication" https://t.co/UVZN4GFFRc #oncology #