Expression of KID syndromic mutation Cx26S17F produces hyperactive hemichannels in supporting cells of the organ of Corti. Original Research Article by Dr. Ana C. Abbott et al. Read more here👇 https://t.co/eP4PUkzyeG #CellDevBio #KIDsyndromicmutation http
Expression of KID syndromic mutation Cx26S17F produces hyperactive hemichannels in supporting cells of the organ of Corti https://t.co/TV1Yj4T9TG
Expression of KID syndromic mutation Cx26S17F produces hyperactive hemichannels in supporting cells of the organ of Corti https://t.co/4pf7TFUOhx
New Research: Expression of KID syndromic mutation Cx26S17F produces hyperactive hemichannels in supporting cells of the organ of Corti: Some mutations in gap junction protein Connexin 26 (Cx26) lead to syndromic deafness, where hearing… https://t.co/u1ha9