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Open Access UCL Research: Synthetic Fragments of Receptor for Advanced Glycation End Products Bind Beta-Amyloid 1-40 and Protect Primary Brain Cells From Beta-Amyloid Toxicity https://t.co/vlWbwfZuiU
Open Access UCL Research: Synthetic Fragments of Receptor for Advanced Glycation End Products Bind Beta-Amyloid 1-40 and Protect Primary Brain Cells From Beta-Amyloid Toxicity https://t.co/vlWbwfZuiU
人工RAGEはβアミロイド1-40に結合し、アミロイドから脳を守る。#AGE https://t.co/dAfxhm5NEc
RT @FrontNeurosci: Synthetic Fragments of Receptor For Advanced Glycation End Products Bind Beta-Amyloid 1-40 And Protect Primary Brain Cel…
RT @FrontNeurosci: Synthetic Fragments of Receptor For Advanced Glycation End Products Bind Beta-Amyloid 1-40 And Protect Primary Brain Cel…