RT @FrontImmunol: New Research: Fcγ Receptor IIB Controls Skin Inflammation in an Active Model of Epidermolysis Bullosa Acquisita: Epidermo…
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New Research: Fcγ Receptor IIB Controls Skin Inflammation in an Active Model of Epidermolysis Bullosa Acquisita: Epidermolysis bullosa acquisita (EBA) is an autoimmune skin blistering disease characterized by IgG autoantibodies (aAb) against… https://t.co/
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Fcγ Receptor IIB Controls Skin Inflammation in an Active Model of Epidermolysis Bullosa Acquisita https://t.co/Xz4cTLpH7H
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RT @RalfLudwig11: New in Research Topic #Skin #Autoimmunity: Fcγ Receptor IIB Controls Skin Inflammation in an Active Model of Epidermolysi…